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- W2085141107 abstract "Neuromyelitis optica (NMO), which includes Devic disease, is widely accepted as a distinct clinical and pathologic entity separate from multiple sclerosis (MS). In NMO, the primary pathologic change is loss of aquaporin 4 water channel protein on astrocytes, occasionally with secondary demyelination.1 NMO is phenotypically similar to optico-spinal MS (OSMS) described in Asian MS populations; particularly in patients with longitudinally extensive transverse myelitis (LETM).2In 2004, Lennon et al.3 reported a characteristic immunofluorescence autoantibody staining pattern of CNS tissues with serum from patients with NMO; IgG deposition occurred around microvessels of the pia, sub-pia, and Virchow-Robin spaces and colocalized with laminin. This autoantibody was named NMO-IgG and was subsequently shown to bind to the predominant CNS water channel aquaporin-4 (AQP4).4 NMO-IgG positivity (and by inference anti-AQP4 antibody positivity) has now been included as one of the three supportive criteria in the recently revised diagnostic criteria for NMO.5 Inclusion of NMO-IgG positivity as part of the diagnostic criteria for NMO has resulted in phenotypic spread. For example, involvement of the brain, previously …" @default.
- W2085141107 created "2016-06-24" @default.
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- W2085141107 date "2008-06-02" @default.
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- W2085141107 title "To test or not to test: NMO-IgG and optic neuritis" @default.
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- W2085141107 doi "https://doi.org/10.1212/01.wnl.0000313843.78606.cd" @default.
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