Matches in SemOpenAlex for { <https://semopenalex.org/work/W2086012224> ?p ?o ?g. }
- W2086012224 endingPage "3752" @default.
- W2086012224 startingPage "3745" @default.
- W2086012224 abstract "Mendelian heritable pain disorders have provided insights into human pain mechanisms and suggested new analgesic drug targets. Interestingly, many of the heritable monogenic pain disorders have been mapped to mutations in genes encoding ion channels. Studies in transgenic mice have also implicated many ion channels in damage sensing and pain modulation. It seems likely that aberrant peripheral or central ion channel activity underlies or initiates many pathological pain conditions. Understanding the mechanistic basis of ion channel malfunction in terms of trafficking, localization, biophysics, and consequences for neurotransmission is a potential route to new pain therapies." @default.
- W2086012224 created "2016-06-24" @default.
- W2086012224 creator A5040885096 @default.
- W2086012224 creator A5071292118 @default.
- W2086012224 creator A5074297075 @default.
- W2086012224 date "2010-11-01" @default.
- W2086012224 modified "2023-10-18" @default.
- W2086012224 title "Pain as a channelopathy" @default.
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