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- W2086338081 abstract "Following a short period of amplitude enhancement, the f2−f1 DPOAE can be suppressed during continuous ipsilateral stimulation with primary tones. Evidence for and against efferent control of such response alterations has been presented. Here, an exacerbation of f2−f1 suppression, apparent following a short rest from continuous stimulation, is described and the hypothesis that it is efferent-mediated is tested. The f2−f1 DPOAE to intermittent or continuous primaries ( f1=6.25 kHz; f2=7.5 kHz; L1=L2=60 dB SPL) was recorded in urethane-anesthetized guinea pigs with sectioned middle ear muscles before, during, and after perfusion of the cochlear perilymph compartment with antagonists of olivocochlear efferent activity (curare, 1 μM; bicuculline, 10 μM) or of action potentials in general (tetrodotoxin, TTX; 1 μM). Results confirmed an initial f2−f1 amplitude enhancement and gradual suppression during continued stimulation. Following a 1-min rest, however, the DPOAE was further suppressed from its pre-rest level. Thereafter, its amplitude increased rapidly, reaching a maximum within 1–2 min. These amplitude alterations were intensity dependent. Moreover, they were reduced, but not blocked by efferent antagonists and were largely unaffected by TTX, suggesting that additional (e.g., local) alterations in cochlear status impact the cochlear mechanical response to continuous sound stimulation. [Work supported by NIH DC00007; DAMD 17-93-V-3013.]" @default.
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- W2086338081 date "1994-05-01" @default.
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- W2086338081 title "A suppressive ‘‘off‐effect’’ in the f2−f1 DPOAE response to continuous, moderate‐level primary stimulation" @default.
- W2086338081 doi "https://doi.org/10.1121/1.409586" @default.
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