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• W2086541404 abstract "HomeHypertensionVol. 53, No. 1Response to Lead, Smoking, and Peripheral Vascular Function Free AccessLetterPDF/EPUBAboutView PDFView EPUBSections ToolsAdd to favoritesDownload citationsTrack citationsPermissions ShareShare onFacebookTwitterLinked InMendeleyReddit Jump toFree AccessLetterPDF/EPUBResponse to Lead, Smoking, and Peripheral Vascular Function Jean François Argacha, Dionysios Adamopoulos and Philippe van de Borne Jean François ArgachaJean François Argacha Department of Cardiology, Erasme Hospital, Université Libre de Bruxelles, Brussels, Belgium Search for more papers by this author , Dionysios AdamopoulosDionysios Adamopoulos Department of Cardiology, Erasme Hospital, Université Libre de Bruxelles, Brussels, Belgium Search for more papers by this author and Philippe van de BornePhilippe van de Borne Department of Cardiology, Erasme Hospital, Université Libre de Bruxelles, Brussels, Belgium Search for more papers by this author Originally published10 Nov 2008https://doi.org/10.1161/HYPERTENSIONAHA.108.121913Hypertension. 2009;53:e6Other version(s) of this articleYou are viewing the most recent version of this article. Previous versions: November 10, 2008: Previous Version 1 Tobacco smoke contains thousands of different compounds, including a mixture of metals such as aluminum, arsenic, cadmium, chromium, copper, lead, mercury, nickel, and zinc. In our study, 1-hour exposure to passive smoking exerted some acute deleterious effects on aortic wave reflection and microvascular function.1 We incriminated the neurohumoral effects of nicotine as a main determinant of the cardiovascular reaction after environmental tobacco smoke exposure. However, a potential hazardous effect of other components (such as metals) contained in the tobacco smoke cannot be ruled out by the present study. In addition, we reported recently in vitro that the acute endothelial toxicity of passive smoking cannot be simply ascribed to a nicotine-dependent mechanism.2We appreciate the letter by Kisters et al,3 and we agree that a nonnicotinic tobacco component, such as metal constituents, may also impair the vascular endothelium. Indeed, metals enhance oxidative stress of cytoskeletal proteins4 and damage endothelial cell gene transcription.5 We also believe that further works are needed to examine the contribution of metal exposure to arteriosclerosis arising from active and passive smoking.Sources of FundingThis study was supported and funded by the Erasme Foundation, AstraZeneca, Pfizer, Medtronic, Novartis, the David and Alice Van Buuren Fund, the Belgian National Fund for Research, the Lambertine Lacroix Prize, the Foundation Saucez-Van Poucke, and the Foundation for Cardiac Surgery. None of the funding sources intervened in any aspects of the study.DisclosuresNone.1 Argacha JF, Adamopoulos D, Gujic M, Fontaine D, Amyai N, Berkenboom G, van de Borne P. Acute effects of passive smoking on peripheral vascular function. Hypertension. 2008; 51: 1506–1511.LinkGoogle Scholar2 Argacha JF, Fontaine D, Adamopoulos D, Ajose A, van de Borne P, Fontaine J, Berkenboom G. Acute effect of sidestream cigarette smoke extract on vascular endothelial function. J Cardiovasc Pharmacol. 2008; 52: 262–267.CrossrefMedlineGoogle Scholar3 Kisters K, Gremmler B, Hausberg M. Lead, smoking, and peripheral vascular function. Hypertension. 2009; 53: e5.LinkGoogle Scholar4 Bernhard D, Csordas A, Henderson B, Rossmann A, Kind M, Wick G. Cigarette smoke metal-catalyzed protein oxidation leads to vascular endothelial cell contraction by depolymerization of microtubules. FASEB J. 2005; 19: 1096–1107.CrossrefMedlineGoogle Scholar5 Bernhard D, Rossmann A, Henderson B, Kind M, Seubert A, Wick G. Increased serum cadmium and strontium levels in young smokers: effects on arterial endothelial cell gene transcription. Arterioscler Thromb Vasc Biol. 2006; 26: 833–838.LinkGoogle Scholar Previous Back to top Next FiguresReferencesRelatedDetails January 2009Vol 53, Issue 1 Advertisement Article InformationMetrics https://doi.org/10.1161/HYPERTENSIONAHA.108.121913 Originally publishedNovember 10, 2008 PDF download Advertisement SubjectsPharmacology" @default.
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