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• W2086559218 abstract "The clinicopathological features of malignant cells are sometimes modified by autologous cytokine production. Inflammatory fibrous histiocytoma (IFH) is characterised by leukocyte infiltration and is a variant of malignant fibrous histiocytoma (MFH). We demonstrated that three MFH cell lines (MF-1, MF-3, and MF-4) have the potential to promote neutrophil chemotaxis and to express mRNA for the cytokines, granulocyte-macrophage colony stimulating factor (GM-CSF) and/or interleukin 8/neutrophil attractant/activation protein 1 (IL-8/NAP-1), both with and without interleukin 1 beta (IL-1 beta) stimulation. MF-1 cells showed the spontaneous production of neutrophil chemotactic activity and the expression of both of GM-CSF and IL-8/NAP-1 mRNA, which was enhanced by exogenous IL-1 beta. In contrast, MF-3 cells showed the expression of GM-CSF and IL-8/NAP-1 mRNA with IL-1 beta stimulation but not without it, and MF-4 cells expressed only IL-8/NAP-1 mRNA when stimulated with IL-1 beta (time- and dose-dependent expression). These findings suggest that neutrophil chemotactic cytokines derived from IFH cells might be responsible for the prominent infiltration of neutrophils in this disease." @default.
• W2086559218 created "2016-06-24" @default.
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• W2086559218 date "1993-03-01" @default.
• W2086559218 modified "2023-10-09" @default.
• W2086559218 title "Neutrophil chemotactic factors produced by malignant fibrous histiocytoma cell lines" @default.
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• W2086559218 doi "https://doi.org/10.1038/bjc.1993.95" @default.
• W2086559218 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1968275" @default.
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