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• W2087776586 abstract "Fragile X syndrome (FXS) mental retardation is caused by loss-of-function mutations in an RNA-binding protein, fragile X mental retardation protein (FMRP). Previous studies in patients or animal models of FXS have identified alterations in dendritic spine structure, as well as synaptic plasticity induced by metabotropic glutamate receptors (mGluRs). The translation of multiple messenger RNA (mRNA) targets of FMRP is regulated by mGluRs at synapses. Here, we incorporate data from several studies into a working model of how FMRP regulates mGluR-stimulated protein synthesis and, in turn, regulates protein synthesis–dependent synaptic plasticity. Understanding the complex functions of FMRP at the synapse will lead to a better understanding of the neurobiological underpinnings of mental retardation." @default.
• W2087776586 created "2016-06-24" @default.
• W2087776586 creator A5038698200 @default.
• W2087776586 creator A5089295487 @default.
• W2087776586 date "2008-02-05" @default.
• W2087776586 modified "2023-10-14" @default.
• W2087776586 title "Metabotropic Glutamate Receptors and Fragile X Mental Retardation Protein: Partners in Translational Regulation at the Synapse" @default.
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• W2087776586 doi "https://doi.org/10.1126/stke.15pe6" @default.
• W2087776586 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/18272470" @default.
• W2087776586 hasPublicationYear "2008" @default.
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