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- W2088107651 endingPage "135" @default.
- W2088107651 startingPage "129" @default.
- W2088107651 abstract "There is a naturally occurring gender difference in immune responses which persists after traumatic injury. Physiological levels of 17β-estradiol (E2) are immunostimulatory, whereas high pregnancy and superphysiological levels are immunosuppressive. In contrast, at all concentrations, testosterone suppresses immune responses. Evidence from this laboratory and others suggest that the gender difference in immune responses after injury is mediated in part by alterations in the circulating levels of gonadal steroid hormones through modulation of production of inflammatory and immunoregulatory cytokines, including interleukin-6 (IL-6). Aberrant production of IL-6 is known to be an important mediator of immunity after injury. Since E2 is a critical regulator of IL-6 production and overall immune function, it suggests gender specific therapies should be considered for the treatment of patients." @default.
- W2088107651 created "2016-06-24" @default.
- W2088107651 creator A5001708025 @default.
- W2088107651 creator A5034398180 @default.
- W2088107651 creator A5055025282 @default.
- W2088107651 date "2002-07-01" @default.
- W2088107651 modified "2023-10-11" @default.
- W2088107651 title "Estrogen regulation of immune responses after injury" @default.
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- W2088107651 doi "https://doi.org/10.1016/s0303-7207(02)00106-5" @default.