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- W2088133087 endingPage "445" @default.
- W2088133087 startingPage "436" @default.
- W2088133087 abstract "Macrophages are essential players in induction and progression of atherosclerotic inflammation. The complexity of macrophage phenotypes was observed in human plaques and atherosclerotic lesions in mouse models of atherosclerosis. Plaque macrophages were shown to exhibit a phenotypic range that is intermediate between two extremes, M1 (pro-inflammatory) and M2 (anti-inflammatory). Indeed, in atherosclerosis, macrophages demonstrate phenotypic plasticity to rapidly adjust to changing microenvironmental conditions. In the plaque, serum lipids, serum lipoproteins and various pro- or anti-inflammatory stimuli such as cytokines, chemokines and small bioactive molecules could greatly influence the macrophage phenotype inducing switch towards more proinflammatory or anti-inflammatory properties. Dynamic plasticity of macrophages is achieved by up-regulation and down-regulation of an overlapping set of transcription factors that drive macrophage polarization. Understanding of mechanisms of macrophage plasticity and resolving functional characteristics of distinct macrophage phenotypes should help in the development of new strategies for treatment of chronic inflammation in atherosclerosis and other cardiovascular diseases." @default.
- W2088133087 created "2016-06-24" @default.
- W2088133087 creator A5009648842 @default.
- W2088133087 creator A5011643232 @default.
- W2088133087 creator A5043145631 @default.
- W2088133087 creator A5060595668 @default.
- W2088133087 creator A5075043020 @default.
- W2088133087 creator A5077715103 @default.
- W2088133087 date "2015-04-01" @default.
- W2088133087 modified "2023-10-18" @default.
- W2088133087 title "RETRACTED: Macrophage phenotypic plasticity in atherosclerosis: The associated features and the peculiarities of the expression of inflammatory genes" @default.
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