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• W2088333650 startingPage "836" @default.
• W2088333650 abstract "Heat shock (HS)/stress proteins (HSP) provide protection from a variety of stresses other than HS, including oxidative stress and mitochondria have been implicated as the target of HS-related protection in stressed cultured cells. Here we investigated whether mitochondria also are targets for the HS-mediated protection in vivo. Sprague Dawley rats were exposed, or not, to HS (41 degrees C, 15 min). After a 21 h recovery period, hearts were excised and perfused with or without H2O2 (0.15 mM). Myocardial mitochondria were then isolated, and their oxygen consumption was analyzed. HS prevented H2O2-induced alterations in state 3 respiration while increasing the expression of Hsp70 and heme oxygenase (HO). Thus, in vivo HS protects rat myocardial mitochondrial respiration against the deleterious effects of oxidative injury, a protection relating to Hsp70 and/or HO and targeting state 3 respiration." @default.
• W2088333650 created "2016-06-24" @default.
• W2088333650 creator A5006175729 @default.
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• W2088333650 creator A5071584034 @default.
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• W2088333650 date "1998-05-01" @default.
• W2088333650 modified "2023-09-26" @default.
• W2088333650 title "In VivoHeat Shock Protects Rat Myocardial Mitochondria" @default.
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• W2088333650 doi "https://doi.org/10.1006/bbrc.1998.8717" @default.
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