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• W2088830811 startingPage "124" @default.
• W2088830811 abstract "Animal cells have been widely used to obtain a wide range of products for human and animal healthcare applications. However, the extreme sensitivity of these cells in respect to changes experienced in their environment is evidenced by the activation of a gene-encoded program known as apoptosis, resulting in their death and destruction. From the bioprocess angle, losses in cell viability bring lower productivities and higher risks of product degradation. Consequently, many research efforts have been devoted to the development of apoptosis protective mechanisms, including the metabolic engineering of apoptosis pathways, that has proven effective in diminishing programmed cell death in a variety of biotechnological relevant cell lines. This review is focused especially in the encouraging initial results obtained with the over-expression of cloned anti-apoptosis genes, from both endogenous and viral origin interfering at mitochondrial and initiator caspases levels." @default.
• W2088830811 created "2016-06-24" @default.
• W2088830811 creator A5020377123 @default.
• W2088830811 creator A5029590918 @default.
• W2088830811 creator A5053876476 @default.
• W2088830811 creator A5077050654 @default.
• W2088830811 date "2003-04-01" @default.
• W2088830811 modified "2023-09-25" @default.
• W2088830811 title "Metabolic engineering of apoptosis in cultured animal cells: implications for the biotechnology industry" @default.
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• W2088830811 doi "https://doi.org/10.1016/s1096-7176(03)00024-7" @default.
• W2088830811 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/12850134" @default.
• W2088830811 hasPublicationYear "2003" @default.
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