FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W2088897584> ?p ?o ?g. }

• W2088897584 endingPage "919" @default.
• W2088897584 startingPage "911" @default.
• W2088897584 abstract "Journal of the American Geriatrics SocietyVolume 39, Issue 9 p. 911-919 Senescence-Accelerated Mouse (SAM): A Novel Murine Model of Accelerated Senescence Toshio Takeda MD, Corresponding Author Toshio Takeda MD Department of Senescence Biology, Chest Disease Research Institute, Kyoto University, Sakyo-ku, Kyoto 606, Japan.Department of Senescence Biology, Chest Disease Research Institute, Kyoto University, Sakyo-ku, Kyoto 606, Japan.Search for more papers by this authorMasanori Hosokawa MD, Masanori Hosokawa MD Department of Senescence Biology, Chest Disease Research Institute, Kyoto University, Sakyo-ku, Kyoto 606, Japan.Search for more papers by this authorKeiichi Higuchi PhD, Keiichi Higuchi PhD Department of Senescence Biology, Chest Disease Research Institute, Kyoto University, Sakyo-ku, Kyoto 606, Japan.Search for more papers by this author Toshio Takeda MD, Corresponding Author Toshio Takeda MD Department of Senescence Biology, Chest Disease Research Institute, Kyoto University, Sakyo-ku, Kyoto 606, Japan.Department of Senescence Biology, Chest Disease Research Institute, Kyoto University, Sakyo-ku, Kyoto 606, Japan.Search for more papers by this authorMasanori Hosokawa MD, Masanori Hosokawa MD Department of Senescence Biology, Chest Disease Research Institute, Kyoto University, Sakyo-ku, Kyoto 606, Japan.Search for more papers by this authorKeiichi Higuchi PhD, Keiichi Higuchi PhD Department of Senescence Biology, Chest Disease Research Institute, Kyoto University, Sakyo-ku, Kyoto 606, Japan.Search for more papers by this author First published: September 1991 https://doi.org/10.1111/j.1532-5415.1991.tb04460.xCitations: 279 This work was supported by a grant-in-aid for scientific research and a grant-in-aid for developmental scientific research from the Ministry of Education, Science and Culture and also by grants from the Ministry of Health and Welfare of Japan. Read the full textAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onEmailFacebookTwitterLinkedInRedditWechat REFERENCES 1 Takeda T, Hosokawa M, Takeshita S et al. A new murine model of accelerated senescence. Mech Ageing Dev 1981; 17: 183. 2 Hosokawa M, Takeshita S, Higuchi K et al. Cataract and other ophthalmic lesions in Senescence Accelerated Mouse (SAM). Morphology and incidence of senescence associated ophthalmic changes in mice. Exp Eye Res 1984; 38: 105. 3 Hosokawa M, Kasai R, Higuchi K et al. Grading score system: A method for evaluation of the degree of senescence in Senescence Accelerated Mouse (SAM). Mech Ageing Dev 1984; 26: 91. 4 Gompertz B. On the nature of the function expressive of the law of human mortality and on a new model of determining life contingencies. Philos Trans R Soc London 1825; 115: 513. 5 Takeshita S, Hosokawa M, Irino M et al. Spontaneous age-associated amyloidosis in Senescence Accelerated Mouse (SAM). Mech Ageing Dev 1982; 20: 13. 6 Shimizu K, Kasai R, Yamamuro T et al. Amyloid deposition in the articular structure of AKR senescent mice. Arth Rheum 1981; 24: 1540. 7 Shimizu K, Ishii M, Yamamuro T et al. Amyloid deposition in intervertebral discs of senescence-accelerated mouse. Arthritis Rheum 1982; 25: 710. 8 Higuchi K, Matsumura A, Honma A et al. Systemic senile amyloid in Senescence-Accelerated Mouse: A unique fibril protein demonstrated in tissues from various organs by the unlabeled immunoperoxidase method. Lab Invest 1983; 48: 231. 9 Sashima M, Satoh M, Suzuki A. Oral senile amyloidosis in senescence accelerated mouse (SAM). J Oral Pathol Med 1990; 19: 318. 10 Naiki H, Higuchi K, Hosokawa M et al. Fluorometric determination of amyloid fibrils in vitro using the fluorescent dye, Thioflavine T. Anal Biochem 1989; 177: 244. 11 Naiki H, Higuchi K, Matsushima K et al. Fluorometric examination of tissue amyloid fibril in murine senile amyloidosis: Use of the fluorescent indicator, Thioflavine T. Lab Invest 1990; 62: 768. 12 Matsumura A, Higuchi K, Shimizu K et al. A novel amyloid fibril protein isolated from senescence accelerated mice. Lab Invest 1982; 47: 270. 13 Yonezu T, Higuchi K, Tsunasawa S et al. High homology is present in the primary structures between murine senile amyloid protein (ASSAM) and human apolipoprotein A-II. FEBS Lett 1986; 203: 149. 14 Higuchi K, Matsumura A, Honma A et al. Age-related changes of serum apoprotein SASSAM, apoprotein A-I and low-density lipoprotein levels in Senescence Accelerated Mouse (SAM). Mech Ageing Dev 1984; 26: 311. 15 Higuchi K, Matsumura A, Hashimoto K et al. Isolation and characterization of senile amyloid-related antigenic substance (SASSAM) from mouse serum. Apo SASSAM is a low molecular weight apoprotein of high density lipoprotein. J Exp Med 1983; 158: 1600. 16 Takeshita S, Higuchi K, Hosokawa M et al. Morphological demonstration of cytoplasmic ASsam -related antigenic substance (CASSAM) by an immunoperoxidase technique. Am J Pathol 1985; 121: 455. 17 Higuchi K, Yonezu T, Kogishi K et al. Purification and characterization of a senile amyloid-related antigenic substance (apo SASSAM) from mouse serum. J Biol Chem 1986; 261: 12834. 18 Kunisada T, Higuchi K, Aota S et al. Molecular cloning and nucleotide sequence of cDNA for murine senile amyloid protein: nucleotide substitutions found in apolipoprotein A-II cDNA of senescence accelerated mouse (SAM). Nucleic Acids Res 1986; 14: 5729. 19 Yonezu T, Toda M, Yamagishi H et al. Structural organization of the senile amyloid protein gene in a strain of Senescence Accelerated Mouse (SAM). Gene 1989; 84: 187. 20 Yonezu T, Tsunasawa S, Higuchi K et al. A molecularpathologic approach to murine senile amyloidosis. Serum precursor-Apo A-II variant (pro5→ gln) presents only an amyloidosis-prone SAMP/1 and -P/2 mice. Lab Invest 1987; 57: 65. 21 Higuchi K, Yonezu T, Tsunasawa S et al. The single proline-glutamine substitution at position 5 enhances the potency of amyloid fibril formation of murine apo A-II. FEBS Lett 1986; 207: 23. 22 Naiki H, Higuchi K, Yonezu T et al. Metabolism of senile amyloid precursor and amyloidogenesis: Age related acceleration of apolipoprotein A-II clearance in Senescence Accelerated Mouse. Am J Pathol 1988; 130: 579. 23 Takeda T, Higuchi K, Hosokawa M. ASSAM amyloidosis is present in aging mice of many strains, not only in SAM. In: T Isobe, S Araki, F Uchino et al, eds. Amyloid and Amyloidosis. New York: Plenum Publishing Corporation, 1988, p 685. 24 Matsushita M, Tsuboyama T, Kasai R et al. Age related changes in bone mass in the Senescence Accelerated Mouse (SAM): SAM-R/3 and SAM-P/6 as new murine models for senile osteoporosis. Am J Pathol 1986; 125: 276. 25 Newton-John HF, Morgan DB. The loss of bone with age, osteoporosis and fractures. Clin Orthop 1970; 71: 229. 26 Nordin BEC. Clinical significance and pathogenesis of osteoporosis. Br Med J 1971; 1: 571. 27 Riggs BL, Melton LJ. Evidence for two distinct syndromes of involutional osteoporosis. Am J Med 1983; 75: 899. 28 Parfitt AM. Age-related structural changes in trabecular and cortical bone: Cellular mechanism and biochemical consequence. Calcif Tissue Int 1984; 36: S123. 29 Tsukada R, Shino A, Tsuda M et al. Bone changes in the Senescence-Accelerated Mouse (SAM) reared under specific pathogen-free (SPF) conditions. J Bone Miner Metab 1986; 4: 121. 30 Tsuboyama T, Takahashi K, Matsushita M et al. Decreased endosteal formation during cortical bone modeling in SAM-P/6 mice with a low peak bone mass. Bone Miner 1989; 7: 1. 31 Kawase M, Tsuda M, Matsuo T. Accelerated bone resorption in Senescence-Accelerated Mouse (SAM-P/6). J Bone Miner Res 1989; 4: 359. 32 Miyamoto M, Kiyota Y, Yamazaki N et al. Age-related changes in learning and memory in the senescence-accelerated mouse (SAM). Physiol Behav 1986; 38: 399. 33 Yagi H, Katoh S, Akiguchi I et al. Age-related deterioration of ability of acquisition in memory and learning in Senescence Accelerated Mouse: SAM-P/8 as an animal model of disturbances in recent memory. Brain Res 1988; 474: 86. 34 Ohta A, Hirano T, Yagi H et al. Behavioral characteristics of the SAM-P/8 strain in Sidman active avoidance task. Brain Res 1989; 498: 195. 35 Shimada A, Ohta A, Akiguchi I et al. The search for an animal model of aging brain. Proceedings of XIth International Congress of Neuropathology. Neuropathology Suppl IV: (in press). 36 Akiguchi I, Akiyama H, Sugiyama H et al. Morphological changes of the brain of Senescence Accelerated Mouse (SAM-P/8). In: T Takeda, T Matsuo, I Akiguchi et al, eds. Proceedings of the first SAM Kyoto Symposium. The council for SAM research. Kyoto: Fuji Printing Company, 1988, p 67. 37 Akiyama H, Kameyama M, Akiguchi I et al. Periodic acid-Schiff (PAS)-positive, granular structures increase in the brain of Senescence Accelerated Mouse (SAM). Acta Neuropathol 1986; 72: 124. 38 Yagi H, Irino M, Matsushita T et al. Spontaneous spongiform degeneration of the brain stem in SAM-P/8 mice, a newly developed memory-deficient strain. J Neuropathol Exp Neurol 1989; 48: 577. 39 Kornblith G, Olds J. Unit activity in brain stem reticular formation of the rat during learning. J Neurophysiol 1973; 36: 489. 40 Gabriel M, Gregg B, Clancy A et al. Brain stem reticular formation neuronal correlates of stimulus significance and behavior during discriminative avoidance conditioning in rabbits. Behav Neurosci 1986; 100: 171. 41 Zhao X-H, Nomura Y. Age-related changes in uptake and release of L-[3 H] noradrenaline in brain slices of senescence accelerated mouse. Int J Dev Neurosci 1990; 8: 267. 42 Kitamura Y, Zhao X-H, Ohnuki T et al. Ligand-binding characteristics of [3 H] QNB, [3 H] prazosin, [3 H] rauwolscine, [3 H] TCP and [3 H] nitrendipine to cerebral cortical and hippocampal membranes of senescent accelerated mouse, Neurosci Lett 1989; 106: 334. 43 Nomura Y, Kitamura Y, Zhao X-H. Glu system in SAM Brain. In: T Kameyama, F Domino, T Nabeshima, eds. NMDA Related Agents: Biochemistry, Pharmacology and Behavior. Ann Arbor, MI: NPP Books (in press). 44 Katsuki H, Ishihara K, Shimada A et al. Age-related deterioration of long-term potentiation in the CA3 and CA1 regions of hippocampal slices from senescence-accelerated mouse. Arch Gerontol Geriatr 1990; 11: 77. 45 Hosokawa M, Ashida Y, Tsuboyama T et al. Cataract in Senescence Accelerated Mouse (SAM) (II). Development of a new strain of mouse with late appearing cataract. Exp Eye Res 1988; 47: 629. 46 Chen WH, Hosokawa M, Tsuboyama T et al. Age-related changes in the temporomandibular joint of the Senescence Accelerated Mouse (SAM): SAM-P/3 as a new murine model of degenerative joint disease. Am J Pathol 1989; 135: 379. 47 Sashima M, Satoh M, Suzuki A. Incisor abnormality of senescence accelerated mouse (SAM). Gerodontology 1987; 6: 145. 48 Mitsuoka A, Hanada K. In vivo immune reactivities in Senescence Accelerated Mice (SAM). In: T Takeda, T Matsuo, I Akiguchi et al, eds. Proceedings of the first SAM Kyoto Symposium. The council for SAM research, Kyoto: Fuji Printing Company, 1988, p 11. 49 Yoshioka H, Yoshida H, Doi T et al. Autoimmune abnormalities in a murine model of accelerated senescence. Clin Exp Immunol 1989; 75: 129. 50 Hosokawa T, Hosono M, Higuchi K et al. Immune responses in newly developed short-lived SAM Mice I. Age-associated early decline in immune activities of cultured spleen cells. Immunology 1987; 62: 4191. 51 Hosokawa T, Hosono M, Hanada K et al. Immune responses in newly developed short-lived SAM mice II. Selectively impaired T helper cell activity in in vitro antibody response. Immunology 1987; 62: 425. 52 Hanada K, Hosono M, Hosokawa T et al. Immune responses in newly developed short-lived SAM mice III. Genetic control of defective helper T cell activity in in vitro primary antibody response. Immunology 1989; 68: 540. 53 Makinodan T, Kay MMB. Age influence on the immune system. Adv Immunol 1980; 291: 287. 54 Wade AD, Szewczuk MR. Aging, idiotype repertoire shift, and compartmentalization of the mucosal-associated lymphoid system. Adv Immunol 1982; 36: 143. 55 Friedman D, Globerson A. Immune reactivity during aging. II. Analysis of the cellular mechanisms involved in the deficient antibody response in old mice. Mech Ageing Dev 1978; 7: 299. 56 Kishimoto S, Tomino S, Mitsuya H et al. Age-related decline in the in vitro and in vivo synthesis of anti-tetanus toxoid antibody humans. J Immunol 1980; 125: 2347. 57 McCay CM, Crowell MF, Maynard LA. The effect of retarded growth upon the length of life span and upon the ultimate body size. J Nutr 1935; 10: 63. 58 Harper AE. Nutrition, aging and longevitiy. Am J Clin Nutr 1982; 36: 737. 59 Kohno A, Yonezu T, Matsushita M et al. Chronic food restriction modulates the advance of senescence in Senescence Accelerated Mouse (SAM). J Nutr 1985; 115: 1259. 60 Umezawa M, Hanada K, Naiki H et al. Effects of dietary restriction on age related immune dysfunction in the Senescence Accelerated Mouse (SAM). J Nutr 1990; 120: 1393. 61 Yagi K, Yoshino K, Komura S et al. Lipid peroxide levels in the Senescence-Accelerated Mouse. J Clin Biochem Nutr 1988; 5: 21. 62 Komura S, Yoshino K, Kondo K et al. Lipid peroxide levels in the skin of the senescence-accelerated mouse. J Clin Biochem Nutr 1988; 5: 255. 63 Nomura Y, Wang B-X, Qi S-B et al. Biochemical changes related to aging in the senescence-accelerated mouse. Exp Gerontol 1989; 24: 49. 64 Yamagishi H, Kunisada T, Takeda T: Amplification of extra-chromosomal small circular DNAs in a murine model of accelerated senescence. Mech Ageing Dev 1985; 29: 101. 65 Nisitani S, Hosokawa M, Sasaki MS et al. Acceleration of chromosome aberrations in senescence accelerated strains of mice. Mutation Res 1990; 237: 221. 66 Okamoto K, Aoki K. Development of a strain of spontaneously hypertensive rats. Jpn Circ J 1963; 27: 283. 67 Watanabe Y. Serial inbreeding of rabbits with hereditary hyperlipidemia (WHHL)-incidence and development of atherosclerosis and xanthoma. Atherosclerosis 1980; 36: 261. 68 Pearce L, Brown WH. Hereditary premature senescence of the rabbit. I. Chronic form: general features. J Exp Med 1960; 111: 485. 69 Pearce L, Brown WH. Hereditary premature senescence of the rabbit. II. Acute form: general features. J Exp Med 1960; 111: 505. 70 Popp DW: Use of congenic mice to study the genetic basis of degenerative disease. Birth Defects: Orig Artic Ser 1978; 14: 261. 71 Cotran RS, Kumar V, Robbins SL. Diseases of Aging. In: Robbins Pathologic Basis of Disease. Philadelphia: W.B. Saunders Company, 1989, p 543. 72 Hosokawa M, Takeda T. Development of Senescence Accelerated Mouse (SAM) and the contribution of this model to studies on aging. In: T Takeda, T Matsuo, I Akiguchi et al, eds. Proceedings of the first SAM Kyoto symposium. Kyoto: Fuji Printing Business Company, 1988, p 1. 73 Masoro EJ. Animal models in Aging Research. In: EL Schneider, JW Rowe, eds. Handbook of the Biology of Aging. San Diego: Academic Press, 1990, p 72. 74 Goldstein S. Human genetic disorders that feature premature onset and accelerated progression of biological aging. In EL Schneider, ed. The Genetics of Aging. New York: Plenum Press, 1978, p 171. 75 Martin GM, Turker MS. Minireview: Model system for the genetic analysis of mechanisms of aging. J Gerontol 1988; 43: B33. Citing Literature Volume39, Issue9September 1991Pages 911-919 ReferencesRelatedInformation" @default.
• W2088897584 created "2016-06-24" @default.
• W2088897584 creator A5039522320 @default.
• W2088897584 creator A5073321511 @default.
• W2088897584 creator A5079047140 @default.
• W2088897584 date "1991-09-01" @default.
• W2088897584 modified "2023-10-04" @default.
• W2088897584 title "Senescence-Accelerated Mouse (SAM): A Novel Murine Model of Accelerated Senescence" @default.
• W2088897584 cites W1488537934 @default.
• W2088897584 cites W1581345242 @default.
• W2088897584 cites W1601260221 @default.
• W2088897584 cites W167981432 @default.
• W2088897584 cites W1753111831 @default.
• W2088897584 cites W1901983160 @default.
• W2088897584 cites W1952062893 @default.
• W2088897584 cites W1968951164 @default.
• W2088897584 cites W1974163627 @default.
• W2088897584 cites W1979103436 @default.
• W2088897584 cites W1979383012 @default.
• W2088897584 cites W1980135713 @default.
• W2088897584 cites W1980219067 @default.
• W2088897584 cites W1987163746 @default.
• W2088897584 cites W1989485117 @default.
• W2088897584 cites W1995247791 @default.
• W2088897584 cites W1998599861 @default.
• W2088897584 cites W1999627990 @default.
• W2088897584 cites W1999920978 @default.
• W2088897584 cites W1999933583 @default.
• W2088897584 cites W2009664223 @default.
• W2088897584 cites W2013451393 @default.
• W2088897584 cites W2015536134 @default.
• W2088897584 cites W2016612223 @default.
• W2088897584 cites W2021940920 @default.
• W2088897584 cites W2023543550 @default.
• W2088897584 cites W2024793190 @default.
• W2088897584 cites W2026787317 @default.
• W2088897584 cites W2029785248 @default.
• W2088897584 cites W2034571171 @default.
• W2088897584 cites W2039799588 @default.
• W2088897584 cites W2044176662 @default.
• W2088897584 cites W2049205278 @default.
• W2088897584 cites W2052403955 @default.
• W2088897584 cites W2053410220 @default.
• W2088897584 cites W2058878434 @default.
• W2088897584 cites W2063593314 @default.
• W2088897584 cites W2064915748 @default.
• W2088897584 cites W2070795813 @default.
• W2088897584 cites W2084177469 @default.
• W2088897584 cites W2084514672 @default.
• W2088897584 cites W2086709176 @default.
• W2088897584 cites W2088038548 @default.
• W2088897584 cites W2088961525 @default.
• W2088897584 cites W2102347865 @default.
• W2088897584 cites W2132504853 @default.
• W2088897584 cites W2159246062 @default.
• W2088897584 cites W2166023743 @default.
• W2088897584 cites W2199758839 @default.
• W2088897584 cites W2221835116 @default.
• W2088897584 cites W2225531383 @default.
• W2088897584 cites W2460295631 @default.
• W2088897584 cites W4242939048 @default.
• W2088897584 doi "https://doi.org/10.1111/j.1532-5415.1991.tb04460.x" @default.
• W2088897584 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/1885867" @default.
• W2088897584 hasPublicationYear "1991" @default.
• W2088897584 type Work @default.
• W2088897584 sameAs 2088897584 @default.
• W2088897584 citedByCount "340" @default.
• W2088897584 countsByYear W20888975842012 @default.
• W2088897584 countsByYear W20888975842013 @default.
• W2088897584 countsByYear W20888975842014 @default.
• W2088897584 countsByYear W20888975842015 @default.
• W2088897584 countsByYear W20888975842016 @default.
• W2088897584 countsByYear W20888975842017 @default.
• W2088897584 countsByYear W20888975842018 @default.
• W2088897584 countsByYear W20888975842019 @default.
• W2088897584 countsByYear W20888975842020 @default.
• W2088897584 countsByYear W20888975842021 @default.
• W2088897584 countsByYear W20888975842022 @default.
• W2088897584 countsByYear W20888975842023 @default.
• W2088897584 crossrefType "journal-article" @default.
• W2088897584 hasAuthorship W2088897584A5039522320 @default.
• W2088897584 hasAuthorship W2088897584A5073321511 @default.
• W2088897584 hasAuthorship W2088897584A5079047140 @default.
• W2088897584 hasConcept C104317684 @default.
• W2088897584 hasConcept C126322002 @default.
• W2088897584 hasConcept C127716648 @default.
• W2088897584 hasConcept C2779523775 @default.
• W2088897584 hasConcept C522857546 @default.
• W2088897584 hasConcept C54355233 @default.
• W2088897584 hasConcept C71924100 @default.
• W2088897584 hasConcept C74909509 @default.
• W2088897584 hasConcept C86803240 @default.
• W2088897584 hasConceptScore W2088897584C104317684 @default.
• W2088897584 hasConceptScore W2088897584C126322002 @default.
• W2088897584 hasConceptScore W2088897584C127716648 @default.
• W2088897584 hasConceptScore W2088897584C2779523775 @default.
• W2088897584 hasConceptScore W2088897584C522857546 @default.
• W2088897584 hasConceptScore W2088897584C54355233 @default.

• next