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• W2088954865 abstract "In June, 2003, a 29-year-old woman had a routine abdominal ultrasound at 12 weeks' gestation, which showed a possible molar pregnancy. Dilation and curettage (D&C) showed a partial hydatidiform mole (figure, A). Flow cytometry showed that the tissue was triploid. We monitored the patient's β-human chorionic gonadotropin (βhCG), which returned to normal (<5 IU/L) in November, 2003. In April, 2004, her βhCG was increased, and an abdominal ultrasound showed a gestational sac. 1 month later, she developed vaginal bleeding; miscarriage was suspected and D&C showed decidua but no fetal tissue. The bleeding continued, her βhCG remained raised, and in June, 2004, a further D&C showed gestational trophoblastic disease. We reviewed the histological specimen, and diagnosed placental site trophoblastic tumour (PSTT) (figure, B). Her βhCG was 1924 IU/L, and a uterine doppler ultrasound showed a 105 mL uterus with a 4 cm vascularised mass extending into the left adnexa and ovary. Full-body CT showed bilateral pulmonary metastases. MRI of the brain showed no abnormalities. We started her on systemic chemotherapy in conjunction with intrathecal methotrexate as prophylaxis for occult central nervous system disease involvement. βhCG returned to normal after 11 weeks of treatment, and the left adnexal and thoracic lesions disappeared. She then had a hysterectomy and left sided salpingo-oophorectomy to debulk any residual tumour. Histopathology showed that the only viable tumour persisted as isolated cells in the left ovary (figure, C)." @default.
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• W2088954865 date "2005-08-01" @default.
• W2088954865 modified "2023-09-25" @default.
• W2088954865 title "Placental site trophoblastic tumour arising from a partial hydatidiform mole" @default.
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• W2088954865 doi "https://doi.org/10.1016/s0140-6736(05)67143-7" @default.
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