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- W2088988576 abstract "The immune system serves to protect organisms from infectious pathogens. During infection, lymphocytes and cells of the innate immune system, expressing receptors that recognize foreign antigens, proliferate and differentiate to develop effector functions that help to kill the pathogens. Effector functions, such as cellular or antibody mediated cytotoxicity, and inflammatory cytokines can be harmful to the host. To limit damage to healthy tissue, mechanisms have evolved to shut down immune responses, including cell inactivation and cell death. Here we discuss recent studies demonstrating that the death of antigen-activated T lymphocytes during termination of an immune response is initiated by the BH3-only Bcl-2 family member Bim and also requires its multi-BH domain pro-apoptotic relatives Bax and Bak." @default.
- W2088988576 created "2016-06-24" @default.
- W2088988576 creator A5046532595 @default.
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- W2088988576 date "2004-11-01" @default.
- W2088988576 modified "2023-10-16" @default.
- W2088988576 title "T-lymphocyte death during shutdown of an immune response" @default.
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- W2088988576 doi "https://doi.org/10.1016/j.it.2004.08.012" @default.
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