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• W2090149706 abstract "Oestrogen exposure has long been considered to be a main risk factor of breast cancer. More recently, interest has also focused on the possible carcinogenic influence from oestrogen metabolites, such as catechol oestrogens. O-methylation, catalysed by Catechol-O-Methyltransferase (COMT), is one pathway by which the potentially carcinogenic catechol oestrogens can be inactivated. The gene coding for COMT protein contains a single-nucleotide polymorphism (SNP), resulting in an amino acid shift Val → Met, which has been shown to determine high- and low-activity configuration of the enzyme. We hypothesized that the low-activity allele, COMTMet, may be implicated in early onset breast cancer. In the present case–control study, including 126 young breast cancer patients (≤ 36 years) and 117 healthy female blood donors, we analysed the association between COMTMet genotype and risk of breast cancer. No significant difference in the frequency of low-/high-activity alleles was found between cases and controls, indicating that the polymorphism, as a single factor, may not contribute to breast carcinogenesis in young women. © 2001 Cancer Research Campaign http://www.bjcancer.com" @default.
• W2090149706 created "2016-06-24" @default.
• W2090149706 creator A5023332828 @default.
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• W2090149706 date "2001-09-01" @default.
• W2090149706 modified "2023-10-16" @default.
• W2090149706 title "Catechol-O-Methyltransferase (COMT) gene polymorphism and breast cancer risk in young women" @default.
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• W2090149706 doi "https://doi.org/10.1054/bjoc.2001.2009" @default.
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