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- W2090635612 abstract "We propose that sleep begins within small groups of highly interconnected neurons and is characterized by altered input → output (i→0) relationships for any specific neuronal group. Further, experimental findings suggest that growth factors, released locally in response to neuronal activity, and acting in paracrine and autocrine fashions, induce the altered i→0 relationships. These growth factors also act to provide the structural basis for synapses. Thus, we envision that sleep mechanisms (neural use-dependent induction of growth factors and their subsequent effects on i→0 relationships) cannot be separated from sleep function (growth factor-induced synaptic sculpturing). This mechanism/firnction is envisioned to take place in all areas of the brain, including sleep regulatory circuits as well as throughout the cortex. Finally, the “sleep” of neuronal groups (altered i→o relationships) is coordinated by the known sleep regulatory circuits and activational-projection systems in the brain. The theory extends and integrates existing sleep theories to cover a broader range of phenomena." @default.
- W2090635612 created "2016-06-24" @default.
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- W2090635612 date "1999-06-01" @default.
- W2090635612 modified "2023-09-27" @default.
- W2090635612 title "Why we sleep: a theoretical view of sleep function" @default.
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- W2090635612 doi "https://doi.org/10.1016/s1087-0792(99)90019-9" @default.
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