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• W2090776305 abstract "It was reported previously that a <i >Candida albicans</i> water-soluble fraction (CAWS), including a mannoprotein and <i >&#x3b2;</i>-glucan complex, has strong potency in inducing fatal necrotizing arteritis in DBA/2 mice. In this study, histopathological changes and cardiac function were investigated in this system. One mg/day of CAWS was given to DBA/2 mice via peritoneal injection for five days. The CAWS-treated DBA/2 mice were induced aortitis and died at an incidence of 100&#x25; within several weeks. Histological findings included stenosis in the left ventricular outflow tract (LVOT) and severe inflammatory changes of the aortic valve with fibrinoid necrosis. Cardiomegaly was observed and heart weight increased 1.62 fold (<svg style=vertical-align:-0.30096pt;width:56.150002px; id=M1 height=11.0625 version=1.1 viewBox=0 0 56.150002 11.0625 width=56.150002 xmlns=http://www.w3.org/2000/svg> <g transform=matrix(1.25,0,0,-1.25,0,11.0625)> <g transform=translate(72,-63.15)> <text transform=matrix(1,0,0,-1,-71.95,63.5)> <tspan style=font-size: 12.50px; x=0 y=0>𝑃</tspan> <tspan style=font-size: 12.50px; x=10.890113 y=0><</tspan> <tspan style=font-size: 12.50px; x=22.930502 y=0>0</tspan> <tspan style=font-size: 12.50px; x=29.182001 y=0>.</tspan> <tspan style=font-size: 12.50px; x=32.307751 y=0>0</tspan> <tspan style=font-size: 12.50px; x=38.559254 y=0>1</tspan> </text> </g> </g> </svg>). Echocardiography revealed a severe reduction in contractility and dilatation of the cavity in the left ventricle (LV): LV fractional shortening (LVFS) decreased from 71&#x25; to 38&#x25; (<svg style=vertical-align:-0.30096pt;width:56.150002px; id=M2 height=11.0625 version=1.1 viewBox=0 0 56.150002 11.0625 width=56.150002 xmlns=http://www.w3.org/2000/svg> <g transform=matrix(1.25,0,0,-1.25,0,11.0625)> <g transform=translate(72,-63.15)> <text transform=matrix(1,0,0,-1,-71.95,63.5)> <tspan style=font-size: 12.50px; x=0 y=0>𝑃</tspan> <tspan style=font-size: 12.50px; x=10.890113 y=0><</tspan> <tspan style=font-size: 12.50px; x=22.930502 y=0>0</tspan> <tspan style=font-size: 12.50px; x=29.182001 y=0>.</tspan> <tspan style=font-size: 12.50px; x=32.307751 y=0>0</tspan> <tspan style=font-size: 12.50px; x=38.559254 y=0>1</tspan> </text> </g> </g> </svg>), and the LV end-diastolic diameter (LVDd) increased from 2.21 mm to 3.26 mm (<svg style=vertical-align:-0.30096pt;width:56.150002px; id=M3 height=11.0625 version=1.1 viewBox=0 0 56.150002 11.0625 width=56.150002 xmlns=http://www.w3.org/2000/svg> <g transform=matrix(1.25,0,0,-1.25,0,11.0625)> <g transform=translate(72,-63.15)> <text transform=matrix(1,0,0,-1,-71.95,63.5)> <tspan style=font-size: 12.50px; x=0 y=0>𝑃</tspan> <tspan style=font-size: 12.50px; x=10.890113 y=0><</tspan> <tspan style=font-size: 12.50px; x=22.930502 y=0>0</tspan> <tspan style=font-size: 12.50px; x=29.182001 y=0>.</tspan> <tspan style=font-size: 12.50px; x=32.307751 y=0>0</tspan> <tspan style=font-size: 12.50px; x=38.559254 y=0>1</tspan> </text> </g> </g> </svg>). The titer of BNP mRNA increased in the CAWS-treated group. Severe inflammatory changes resulting from CAWS brought about lethal LV dysfunction by aortic valve deformation with LVOT stenosis. This system is proposed as an easy and useful experimental model of heart failure because CAWS arteritis can be induced by CAWS injection alone." @default.
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• W2090776305 date "2012-01-01" @default.
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• W2090776305 title "A Model of Left Ventricular Dysfunction Complicated by CAWS Arteritis in DBA/2 Mice" @default.
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• W2090776305 doi "https://doi.org/10.1155/2012/570297" @default.
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