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- W2090816124 abstract "A previous study in this laboratory has shown that dietary copper (Cu) status mediates the development of thrombotic lesions in female mice. In the present investigation, groups (n = 16) of 6-week-old male and female Swiss-Webster mice were fed Cu-supplemented (9.0 mg Cu/kg) or Cu-deficient (0.2 mg Cu/kg) diets and deionized water for 49–53 days. Thrombotic lesions were observed exclusively in Cu-deficient female mice. Mice fed the Cu-deficient, compared with Cu-supplemented, diet were found to have significantly increased activated partial thromboplastin time (APTT; P < 0.001) and prothrombin time (PT; P < 0.01). Significantly decreased plasma coagulation factor V (P < 0.01) and factor VIII (P < 0.0001) activities were observed in Cu-deficient mice. APTT was significantly (P < 0.01) increased while factor VIII activity was significantly (P < 0.001) decreased in female, compared with male, mice. A significant (P < 0.05) Cu × sex interaction was found for APTT. Although the mechanism involved in the development of thrombotic lesions in Cu-deficient female mice during this study is presently unclear, these results clearly demonstrate that hemostatic function may be mediated by dietary Cu status." @default.
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- W2090816124 date "1992-08-01" @default.
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- W2090816124 title "Effects of a dietary copper deficiency on plasma coagulation factor activities in male and female mice" @default.
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- W2090816124 doi "https://doi.org/10.1016/0955-2863(92)90012-8" @default.
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