Matches in SemOpenAlex for { <https://semopenalex.org/work/W2090870107> ?p ?o ?g. }
- W2090870107 endingPage "24" @default.
- W2090870107 startingPage "13" @default.
- W2090870107 abstract "The accumulation of genetic damage in the forms of activated proto-oncogenes and inactivated tumor-suppressor genes is the driving force in the evolution of a normal cell to a malignant cell. For example, both the activation of ras oncogenes and the inactivation of several suppressor genes, including p53, have been observed in the development of human colon and lung tumors. Point mutations in key codons can activate ras proto-oncogenes and inactivate the p53 suppressor gene. Thus, several critical genes for tumorigenesis are potential targets for carcinogens and radiation that can induce point mutations at low doses. The ras proto-oncogenes are targets for many genotoxic carcinogens. Activation of the ras gene is an early event--probably the" @default.
- W2090870107 created "2016-06-24" @default.
- W2090870107 creator A5012903048 @default.
- W2090870107 creator A5031224691 @default.
- W2090870107 creator A5047497347 @default.
- W2090870107 creator A5072228402 @default.
- W2090870107 date "1992-11-01" @default.
- W2090870107 modified "2023-10-14" @default.
- W2090870107 title "Role of proto-oncogene activation in carcinogenesis." @default.
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