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- W2090885069 abstract "Key Points HSPCs are recruited to S aureus–infected skin wounds, differentiate into neutrophils, and hasten resolution of infection. Bacterial sensing via TLR2 elicits PGE2 production in HSPCs that provides autocrine feedback to meet the demand for local granulopoiesis." @default.
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- W2090885069 date "2013-09-05" @default.
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- W2090885069 title "Staphylococcus aureus recognition by hematopoietic stem and progenitor cells via TLR2/MyD88/PGE2 stimulates granulopoiesis in wounds" @default.
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- W2090885069 doi "https://doi.org/10.1182/blood-2012-11-466268" @default.
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