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- W2091051168 endingPage "646" @default.
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- W2091051168 abstract "Epidemiological studies have provided overwhelming evidence for a causal role of chronic HBV infection in the development of hepatocellular carcinoma (HCC), but the molecular mechanisms underlying virally-induced tumorigenesis remain largely debated. In the absence of a dominant oncogene encoded by the HBV genome, indirect roles have been proposed, including insertional activation of cellular oncogenes by HBV DNA integration, induction of genetic instability by viral integration or by the regulatory protein HBx, and long term effects of viral proteins in enhancing immune-mediated liver disease. In this chapter, we discuss different models of HBV-mediated liver cell transformation based on animal systems of hepadnavirus infection as well as functional studies in hepatocyte and hepatoma cell lines. These studies might help identifying the cellular effectors connecting HBV infection and liver cell transformation." @default.
- W2091051168 created "2016-06-24" @default.
- W2091051168 creator A5023936848 @default.
- W2091051168 creator A5040888090 @default.
- W2091051168 creator A5088504138 @default.
- W2091051168 creator A5091558539 @default.
- W2091051168 date "2009-10-30" @default.
- W2091051168 modified "2023-10-16" @default.
- W2091051168 title "Liver Cell Transformation in Chronic HBV Infection" @default.
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