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• W2091125036 abstract "Bronchiolectasis is a manifestation of chronic bronchitis characterized by saccular dilatation of the terminal bronchioles. The more proximal bronchi may or may not show radiological changes. Zorini and Pigorini (1) first recorded the condition as “larval bronchiectasis” in 1934; in 1951 they reported that they had a personal experience with 100 cases. They recorded the clinical picture as one of chronic bronchitis in which hemoptysis is a prominent symptom. The chest films were negative, but the bronchograms showed small clubshaped or rosary-shaped distal branches. In the single case recorded by these writers symptoms were progressive and surgery was without apparent benefit. Simon and Galbraith (2), in 1953, described the bronchographic findings in 90 patients with a clinical diagnosis of chronic bronchitis. The bronchographic observations consisted in dilatation, diverticulosis, and narrowing of the bronchi and pooling of the contrast medium in the terminal bronchioles. Peripheral pooling occurred in 26 cases, mainly in those with clinical evidence of emphysema. The pathological changes in chronic bronchitis were reported by Reid (3) in 1954. The early cases were said to show hypertrophy of the mucus-secreting elements, and the more advanced disease, a purulent bronchiolitis. There were abscess cavities, obliteration of the bronchiolar lumen, and dilatation of the bronchioles which was diffuse or localized, with or without stenosis, partial or complete. Some cases were acute, while others were chronic, with scarring and compensatory volume changes in the alveoli. By injecting red lead into 5 of the specimens which showed peripheral pooling (Fig. 1), Reid (4) found that the lobulated bronchioles measured 3 to 5 mm. and arose from bronchioles measuring 1 mm. in diameter. The walls of these spaces were usually lined with respiratory epithelium and contained muscle and fibers confirming a bronchiolar origin. Sometimes the epithelium rested on a fibrous base, suggesting that, although distention is probably the most important factor, ulceration may also play a part in the production of the deformities. Case Reports Case I: F. B., a 39-year-old white automobile mechanic, complained chiefly of chest pain. About ten years earlier, he first began to have recurrent winter colds, characterized by coryza and cough. In 1951, 1952, and 1954, he had pneumonia. Fever, cough, chest pain, and dyspnea developed in November 1950, and about Dec. 6 the patient was admitted to Norfolk General Hospital, where he remained eleven days. During his hospital stay he had vague aches and pains in the chest and coughed up some tenacious mucopurulent sputum. There was no hemoptysis, fever, chills, or night sweats. The family history was noncontributory. Physical examination was negative except for some fine crackling or moist rãles in both lung bases. The white blood count ranged from 12,000 to 16,000, with an increase of polymorphonuclear leukocytes." @default.
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• W2091125036 date "1958-06-01" @default.
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• W2091125036 title "Bronchiolectasis in Chronic Bronchitis" @default.
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• W2091125036 doi "https://doi.org/10.1148/70.6.848" @default.
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