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• W2091237743 abstract "NO or its derivatives (reactive nitrogen species: RNS) have three types of actions on mitochondria: 1) reversible inhibition of mitochondrial respiration at cytochrome oxidase by NO, and irreversible inhibition at multiple sites by RNS; 2) stimulation of mitochondrial production of superoxide, hydrogen peroxide, and peroxynitrite by NO; and 3) induction of mitochondrial permeability transition (MPT) by RNS. Similarly there are three main roles of mitochondria in NO-induced cell death: a) NO inhibition of respiration can induce necrosis (or excitotoxicity in neurons) and inhibit apoptosis if glycolysis is insufficient to compensate, b) RNS- or oxidant-induced signal transduction or DNA damage may activate the mitochondrial pathway to apoptosis, and c) RNS-induced MPT may induce apoptosis or necrosis." @default.
• W2091237743 created "2016-06-24" @default.
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• W2091237743 date "2001-09-01" @default.
• W2091237743 modified "2023-10-09" @default.
• W2091237743 title "Nitric Oxide, Mitochondria, and Cell Death" @default.
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• W2091237743 doi "https://doi.org/10.1080/15216540152845993" @default.
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