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• W2091253504 endingPage "895" @default.
• W2091253504 startingPage "891" @default.
• W2091253504 abstract "Galanin, a 29-amino acid peptide widely distributed in the central and peripheral nervous systems, was found to induce a concentration-dependent increase in corticosterone secretion and cyclic-AMP release by dispersed rat inner adrenocortical cells (maximal effective concentration, 10(-7) M). The effect of 10(-7) M galanin was blocked by 10(-6) M galantide, a specific antagonist of galanin receptors. Galanin (10(-7) M) also enhanced corticosterone and cyclic-AMP responses of dispersed cells to submaximal but not maximal (10(-9) M) effective concentrations of ACTH, and again this effect was reversed by galantide. The ACTH-receptor antagonist corticotropin-inhibiting peptide (10(-6) M) blocked corticosterone response of dispersed cells to 10(-9) M ACTH but not to 10(-7) M galanin; conversely, the specific protein kinase A inhibitor H-89 (10(-5) M) annulled the secretory response to both ACTH and galanin. In light of these findings, we conclude that galanin stimulates adrenal glucocorticoid secretion in rats, acting through specific receptors, coupled, like those of ACTH, with the adenylate cyclase/protein kinase A-dependent signaling pathway." @default.
• W2091253504 created "2016-06-24" @default.
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• W2091253504 date "1998-05-01" @default.
• W2091253504 modified "2023-09-23" @default.
• W2091253504 title "Galanin Stimulates Glucocorticoid Secretion in Rats through a Receptor-dependent Activation of the Adenylate Cyclase/Protein Kinase A-dependent Signaling Pathway" @default.
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• W2091253504 doi "https://doi.org/10.1016/s0196-9781(98)00028-x" @default.
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• W2091253504 hasPublicationYear "1998" @default.
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