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• W2092080776 startingPage "e105113" @default.
• W2092080776 abstract "ALKBH4, an AlkB homologue in the 2-oxoglutarate and Fe2+ dependent hydroxylase family, has previously been shown to regulate the level of monomethylated lysine-84 in actin and thereby indirectly influences the ability of non-muscular myosin II to bind actin filaments. ALKBH4 modulates fundamental processes including cytokinesis and cell motility, and its depletion is lethal during early preimplantation embryo stage. The aim of this study was to investigate the effect of ALKBH4 deficiency in a physiological context, using inducible Alkbh4 knockout mice. Here, we report that ALKBH4 is essential for the development of spermatocytes during the prophase of meiosis, and that ALKBH4 depletion leads to insufficient establishment of the synaptonemal complex. We also show that ALKBH4 is localized in nucleolar structures of Sertoli cells, spermatogonia and primary spermatocytes." @default.
• W2092080776 created "2016-06-24" @default.
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• W2092080776 date "2014-08-25" @default.
• W2092080776 modified "2023-10-01" @default.
• W2092080776 title "ALKBH4 Depletion in Mice Leads to Spermatogenic Defects" @default.
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• W2092080776 doi "https://doi.org/10.1371/journal.pone.0105113" @default.
• W2092080776 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4143218" @default.
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• W2092080776 hasPublicationYear "2014" @default.
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