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• W2092919436 abstract "Porphyromonas gingivalis strain 381 lipid A showed lower activity in inducing interleukin (IL)-1alpha and IL-1beta production and cytokine mRNA expression than synthetic Escherichia coli lipid A (compound 506) in alveolar macrophages of C57BL/6 mice. Both the lipid As induced tumor necrosis factor alpha in alveolar macrophages and IL-6 in peritoneal macrophages. A calmodulin (CaM) antagonist, W-7, inhibited IL-1beta production and its mRNA expression induced by P. gingivalis lipid A but not compound 506 in alveolar macrophages. A CaM kinase activator reduced the induction of IL-1beta in the serum of mice when administered with compound 506, and protected the mice against the lethal toxicity. The modulation of a variety of intracellular enzymes including the CaM kinase may result in clinical control of endotoxic sepsis." @default.
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• W2092919436 date "2000-03-01" @default.
• W2092919436 modified "2023-10-17" @default.
• W2092919436 title "Prevention of endotoxin-induced lethality in mice by calmodulin kinase activator" @default.
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• W2092919436 doi "https://doi.org/10.1111/j.1574-695x.2000.tb01431.x" @default.
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