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- W2093342212 abstract "Xenoestrogens can affect the healthy functioning of a variety of tissues by acting as potent estrogens via nongenomic signaling pathways or by interfering with those actions of multiple physiological estrogens. Collectively, our and other studies have compared a wide range of estrogenic compounds, including some closely structurally related subgroups. The estrogens that have been studied include environmental contaminants of different subclasses, dietary estrogens, and several prominent physiological metabolites. By comparing the nongenomic signaling and functional responses to these compounds, we have begun to address the structural requirements for their actions through membrane estrogen receptors in the pituitary, in comparison to other tissues, and to gain insights into their typical non-monotonic dose-response behavior. Their multiple inputs into cellular signaling begin processes that eventually integrate at the level of mitogen-activated protein kinase activities to coordinately regulate broad cellular destinies, such as proliferation, apoptosis, or differentiation." @default.
- W2093342212 created "2016-06-24" @default.
- W2093342212 creator A5003738161 @default.
- W2093342212 creator A5069992695 @default.
- W2093342212 creator A5084182459 @default.
- W2093342212 date "2009-12-02" @default.
- W2093342212 modified "2023-10-10" @default.
- W2093342212 title "Nongenomic Signaling Pathways of Estrogen Toxicity" @default.
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- W2093342212 doi "https://doi.org/10.1093/toxsci/kfp288" @default.
- W2093342212 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2902922" @default.
- W2093342212 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/19955490" @default.
- W2093342212 hasPublicationYear "2009" @default.
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