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- W2093817232 abstract "Celiac disease is an intestinal disease caused by intolerance for gluten, a common protein in food. A life-long gluten-free diet is the only available treatment. As it is well established that the interaction between proline-rich gluten derived peptides and the human HLA-DQ2 molecules induces immune responses that lead to disease development, we have now designed a series of gluten peptides in which proline residues were replaced by azidoprolines. These peptides were found to bind to HLA-DQ2 with an affinity similar to that of the natural gluten peptide. Moreover, some of these peptides were found to be non-immunogenic and block gluten induced immune responses. These can thus serve as lead compounds for the development of HLA-DQ2 blocker peptides." @default.
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- W2093817232 date "2008-02-15" @default.
- W2093817232 modified "2023-10-17" @default.
- W2093817232 title "Design of azidoproline containing gluten peptides to suppress CD4+ T-cell responses associated with Celiac disease" @default.
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- W2093817232 doi "https://doi.org/10.1016/j.bmc.2007.10.091" @default.
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