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- W2093990520 abstract "Membrane potential, measured intracellularly, and tension development were recorded in preparations of guinea pig portal vein. Iproveratril (verapamil) (10−5 mole/liter) suppresses all components of spontaneous electrical and mechanical activity and leads to a depolarization of about 15–20 mV. Of the noradrenaline (NA)-induced effects on membrane potential, iproveratril selectively blocks the spike component, leaving the depolarization unchanged. Spike discharges, stimulated by a K+ depolarization (120 mmole/liter), are also blocked by iproveratril, while the K+ depolarization itself remains unaltered. Tension development elicited by NA application is reduced to about 40% of the control by iproveratril, whereas K+-induced contractions are completely abolished. The iproveratril-resistant part of the NA contraction can also be elicited during K+ depolarization, without significant changes of membrane potential. It may be concluded that the response to NA of portal vein smooth muscle consists of two different components." @default.
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- W2093990520 date "1973-01-01" @default.
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- W2093990520 title "Membrane potential and contraction of vascular smooth muscle (portal vein) during application of noradrenaline and high potassium, and selective inhibitory effects of iproveratril (verapamil)" @default.
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- W2093990520 doi "https://doi.org/10.1016/s0026-2862(73)80007-x" @default.
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