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- W2094022211 abstract "<h3>Introduction</h3> The suggestion that antibodies to platelets are responsible for the thrombocytopenia of idiopathic thrombocytopenic purpura (ITP) is based on three observations: (<i>a</i>) infants born of mothers with ITP sometimes exhibit thrombocytopenia<sup>1</sup>; (<i>b</i>) normal volunteers who receive infusions of plasma from patients with ITP sometimes develop thrombocytopenia<sup>2</sup>; (<i>c</i>) transfused platelets do not circulate as long in patients with ITP as in normal recipients.<sup>3</sup>These phenomena indicate that a constituent of the blood of some patients with ITP is able to depress platelet levels of normal persons. These observations, however, are not proof of an antigen-antibody reaction, and it is not known whether this platelet-depressing activity is, in fact, capable of affecting the patient's own platelets. Platelet antibodies have been demonstrated in patients with thrombocytopenia induced by drugs, such as allyl-isopropyl-acetyl-carbamide (Sedormid),<sup>4</sup>quinidine,<sup>5</sup>quinine,<sup>6</sup>and novobiocin<sup>7</sup>; and in patients who have received transfusions.<sup>8-10</sup>" @default.
- W2094022211 created "2016-06-24" @default.
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- W2094022211 date "1962-02-01" @default.
- W2094022211 modified "2023-10-18" @default.
- W2094022211 title "Evaluation of Platelet Antibodies in Idiopathic Thrombocytopenic Purpura" @default.
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- W2094022211 doi "https://doi.org/10.1001/archinte.1962.03620140029006" @default.
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