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- W2094359712 abstract "Myocardial hypoxia is known to be accomapanied by the release of atrial natriuretic factor (ANF), a peptide which dilates the coronary vessels by stimulating particulate guanylyl cyclase. We have assessed whether ANF plays a paracrine role in hypoxic coronary vasodilatation, a reaction which we had previously found to be associated with increased cyclic GMP production. Compound HS 142-1 (100 μg/ml), a specific antagonist of the guanylyl cyclase ANF receptor, inhibited by 50–70% the coronary-vasodilating effects of human ANF (1–10 μg) administered to isolated guinea pig hearts, but affected neither hypoxic coronary vasodilatation nor cyclic GMP overflow. In contrast, the nitric oxide synthase inhibitor Nω-methyl-L-arginine (300 μM) reduced hypoxic coronary vasodilatation and cyclic GMP overproduction by ≈ 70% and 50–60%, respectively. Thus, unlike nitric oxide, ANF appears not to play a paracrine role in hypoxic coronary vasodilatation." @default.
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- W2094359712 date "1993-04-01" @default.
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- W2094359712 title "Accelerated secretion of atrial natriuretic peptide during exercise in effort angina" @default.
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- W2094359712 doi "https://doi.org/10.1016/0002-8703(93)90148-3" @default.
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