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• W2094366129 abstract "One reason for the poor immunogenicity of many tumors may be that they cannot provide signals for CD28-mediated costimulation necessary to fully activate T cells. It has recently become apparent that CTLA-4, a second counterreceptor for the B7 family of costimulatory molecules, is a negative regulator of T cell activation. Here, in vivo administration of antibodies to CTLA-4 resulted in the rejection of tumors, including preestablished tumors. Furthermore, this rejection resulted in immunity to a secondary exposure to tumor cells. These results suggest that blockade of the inhibitory effects of CTLA-4 can allow for, and potentiate, effective immune responses against tumor cells." @default.
• W2094366129 created "2016-06-24" @default.
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• W2094366129 date "1996-03-22" @default.
• W2094366129 modified "2023-10-10" @default.
• W2094366129 title "Enhancement of Antitumor Immunity by CTLA-4 Blockade" @default.
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• W2094366129 doi "https://doi.org/10.1126/science.271.5256.1734" @default.
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