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• W2094828146 abstract "Inflammation is a key component of athero­sclerosis. Genes coding for inflammatory or anti‑inflammatory molecules are considered good candidates for estimating the risk of developing atherosclerosis. Cyclin‑dependent kinase 9 (CDK9), the kinase of positive transcription elongation factor b (P‑TEFb), is crucial in the cell cycle and apoptosis. Previous studies have focused on its inhibition of immune cells for the resolution of inflammation. Considering the effects of inflammation in the pathogenicity of atherosclerosis, decreasing inflammation through the inhibition of CDK9 may be useful for the prognosis of atherosclerosis. The aim of this review was to examine whether inhibition of the CDK9 monocyte may affect the process of inflammation by acting on the cytokine secretion and interacting with endothelial cells (ECs). Thus, CDK9 may be a novel target for the diagnosis and therapy of atherosclerosis." @default.
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• W2094828146 date "2014-07-31" @default.
• W2094828146 modified "2023-09-30" @default.
• W2094828146 title "Cyclin-dependent kinase 9 may as a novel target in downregulating the atherosclerosis inflammation (Review)" @default.
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• W2094828146 doi "https://doi.org/10.3892/br.2014.322" @default.
• W2094828146 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4179722" @default.
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