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• W2094951310 abstract "Objectives YKL-40 is secreted by macrophages, including those in atherosclerotic plaques, neutrophils, and vascular smooth muscle cells. Circulating YKL-40 is elevated in patients with inflammation and increased tissue remodeling. The aim was to examine the sequential changes in serum YKL-40 in patients with acute myocardial infarction (AMI), with and without thrombolytic therapy, as compared with patients with stable coronary artery disease (CAD). Methods YKL-40 was measured by radioimmunoassay in serum from 63 patients. A total of 47 patients had their first AMI [30 with ST segment elevation myocardial infarction (STEMI) were thrombolyzed, 17 with non-STEMI were not thrombolyzed] and 16 patients had CAD. Results Serum YKL-40 at the time of admission was higher in patients with AMI (median: 156 μg/l, range: 40–3000 μg/l) than in patients with CAD (median: 106 μg/l, range: 54–300 μg/l, P=0.048) and healthy participants (median: 102 μg/l, range: 38–514 μg/l, P<0.001). No difference in serum YKL-40 between CAD patients and healthy participants (P=0.89) was observed. No difference in serum YKL-40 between the AMI patients with or without ST-elevations (P=0.12) was observed. The maximum serum YKL-40 during the first 24 h after admission was higher in thrombolyzed STEMI patients than in the nonthrombolyzed, non-STEMI patients (P=0.01) and the CAD patients (P<0.0001). Serum YKL-40 declined consistently from the maximum value just after the AMI and during follow-up. Serum YKL-40 at 90, 180, and 360 days after AMI were significantly higher in nonthrombolyzed than in thrombolyzed patients (P=0.004, P=0.008, P=0.017, respectively). Conclusion These results demonstrated that serum concentrations of YKL-40 are greatly increased in AMI patients with and without thrombolytic therapy." @default.
• W2094951310 created "2016-06-24" @default.
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• W2094951310 date "2008-06-01" @default.
• W2094951310 modified "2023-09-25" @default.
• W2094951310 title "Serum levels of YKL-40 increases in patients with acute myocardial infarction" @default.
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• W2094951310 doi "https://doi.org/10.1097/mca.0b013e3282f40dd5" @default.
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