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- W2095485008 abstract "Deoxyribonucleic acid repair synthesis was studied in one wild-type and two mutant strains of Bacillus subtilis that are defective in excision of pyrimidine dimers. The cells were irradiated with ultraviolet light, and 6-(p-hydroxyphenyl-azo)-uracil was used to block replicative synthesis, allowing only repair synthesis. One of the mutations (uvs-42) resulted in a severe inhibition of incision, dimer excision, and repair synthesis. In contrast, the other mutant (uvr-1) slowly incised and excised dimers and did repair synthesis in patches which appear to be several-fold longer than those in the wild-type strain, apparently because large gaps are produced at excision sites. The results indicate that the primary defect in uvs-42 cells is in initiation of dimer excision, whereas the uvr-1 mutation appears to be a defect in the exonuclease normally used to complete dimer excision." @default.
- W2095485008 created "2016-06-24" @default.
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- W2095485008 date "1979-07-01" @default.
- W2095485008 modified "2023-10-17" @default.
- W2095485008 title "Gap-filling repair synthesis induced by ultraviolet light in a Bacillus subtilis Uvr- mutant" @default.
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- W2095485008 doi "https://doi.org/10.1128/jb.139.1.239-246.1979" @default.
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