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• W2096167220 abstract "The central amygdala is a critical brain region for many aspects of alcohol dependence. Much of the work examining the mechanisms by which the central amygdala mediates the development of alcohol dependence has focused on the interaction of acute and chronic ethanol with central amygdala corticotropin releasing factor signaling. This work has led to a great deal of success in furthering the general understanding of central amygdala neurocircuitry and its role in alcohol dependence. Much of this work has primarily focused on the hypothesis that ethanol utilizes endogenous corticotropin releasing factor signaling to upregulate inhibitory GABAergic transmission in the central amygdala. Work that is more recent suggests that corticotropin releasing factor also plays an important role in mediating anxiety-like behaviors via the enhancement of central amygdala glutamatergic transmission, implying that ethanol/corticotropin releasing factor interactions may modulate excitatory neurotransmission in this brain region. In addition, a number of studies utilizing optogenetic strategies or transgenic mouse lines have begun to examine specific central amygdala neurocircuit dynamics and neuronal subpopulations to better understand overall central amygdala neurocircuitry and the role of neuronal subtypes in mediating anxiety-like behaviors. This review will provide a brief update on this literature and describe some potential future directions that may be important for the development of better treatments for alcohol addiction." @default.
• W2096167220 created "2016-06-24" @default.
• W2096167220 creator A5016281292 @default.
• W2096167220 creator A5091103521 @default.
• W2096167220 date "2015-05-01" @default.
• W2096167220 modified "2023-10-18" @default.
• W2096167220 title "Ethanol and corticotropin releasing factor receptor modulation of central amygdala neurocircuitry: An update and future directions" @default.
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• W2096167220 doi "https://doi.org/10.1016/j.alcohol.2015.01.006" @default.
• W2096167220 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4414799" @default.
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