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- W2096306941 abstract "The thymus is the primary site of T-cell repertoire arousal and induction of tolerance to “self” molecules, as well as major histocompatibility complex (MHC)-restriction. Neonatal thymectomy or congenital absence of the thymus (as in the mutant nude mouse or in patients presenting Di George’s syndrome) induces a profound failure of T-cell development, while B-cell maturation and functions are intact. In humans, however, neonatal thymectomy does not produce massive T-cell depletion, because the thymus exerts its full properties during fetal development. Recently, the question of an extra-thymic T-cell differentiation also has been addressed. Before the characterization of its fundamental immunological properties, the thymus was considered an endocrine gland, and intensive research was conducted with the objective of isolating thymus hormones (1,2)." @default.
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- W2096306941 date "1990-01-01" @default.
- W2096306941 modified "2023-10-17" @default.
- W2096306941 title "Cellular and Molecular Aspects of the Neuroendocrine-Immune Dialogue in T-Cell Differentiation" @default.
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- W2096306941 doi "https://doi.org/10.1007/978-1-4612-3446-3_3" @default.
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