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- W2096422139 abstract "Cyclooxygenase-2 (COX-2) is a highly inducible enzyme exerting diverse actions on cell functions, including proliferation, migration, and DNA damage. Enhanced COX-2 expression may be protective, but excessive expression may be harmful, causing inflammation, atheromatous plaque instability, and intimal hyperplasia. COX-2 transcriptional activation by proinflammatory mediators has been extensively characterized. In this review, the role of C/EBP in regulating COX-2 transcription is highlighted. Recent advances in control of COX-2 transcription by aspirin and salicylate and by a cell cycle-dependent endogenous mechanism are described. The recent progress sheds light on the pathophysiological mechanisms of COX-2 and new transcription-based strategy for controlling COX-2 overexpression and COX-2-mediated cardiovascular diseases." @default.
- W2096422139 created "2016-06-24" @default.
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- W2096422139 date "2005-04-01" @default.
- W2096422139 modified "2023-10-16" @default.
- W2096422139 title "Transcriptional Control of COX-2 via C/EBPβ" @default.
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- W2096422139 doi "https://doi.org/10.1161/01.atv.0000157899.35660.61" @default.
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