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- W2097113872 abstract "Peroxisome proliferator-activated receptors (PPARs) and retinoic acid receptors (RARs), members of the nuclear receptor superfamily, are transcription factors that regulate a variety of important cellular functions. PPARs form heterodimers retinoid X receptor (RXR), an obligate heterodimeric partner for other nuclear receptors. Several novel links between retinoid metabolism and PPAR responses have been identified, and activation of PPAR/RXR expression has been shown to increase response to retinoids. PPAR γ has emerged as a key regulator of cell growth and survival, whose activity is modulated by a number of synthetic and natural ligands. While clinical trials in cancer patients with thiazolidinediones (TZD) have been disappointing, novel structurally different PPAR γ ligands, including triterpenoids, have entered clinical arena as therapeutic agents for epithelial and hematopoietic malignancies. Here we shall review the antitumor advances of PPAR γ , alone and in combination with RAR α ligands in control of cell proliferation, differentiation, and apoptosis and their potential therapeutic applications in hematological malignancies." @default.
- W2097113872 created "2016-06-24" @default.
- W2097113872 creator A5004888325 @default.
- W2097113872 creator A5010974644 @default.
- W2097113872 creator A5022647201 @default.
- W2097113872 creator A5039058444 @default.
- W2097113872 date "2012-01-01" @default.
- W2097113872 modified "2023-09-30" @default.
- W2097113872 title "Effects of PPAR<i>γ</i>Ligands on Leukemia" @default.
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- W2097113872 doi "https://doi.org/10.1155/2012/483656" @default.
- W2097113872 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3364693" @default.
- W2097113872 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/22685453" @default.
- W2097113872 hasPublicationYear "2012" @default.
- W2097113872 type Work @default.