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- W2097383660 endingPage "71" @default.
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- W2097383660 abstract "Novel agents to reduce angiogenesis by targeting vascular endothelial growth factor and other proangiogenic signaling pathways are being developed for advanced nonsmall cell lung cancer. Antibody-based therapies (e.g., aflibercept) and multitargeted tyrosine kinase inhibitors (e.g., sorafenib, sunitinib, and BIBF 1120) are being evaluated in phase III clinical trials. Some antiangiogenic agents have demonstrated distinct profiles in producing a variety of nonhematologic toxicities, including bleeding/hemorrhage, venous and arterial thromboembolic events, gastrointestinal perforation, hypertension, and proteinuria. Elucidating the molecular basis of these toxicities may lead to clinical benefits by improving patient selection and allowing for the development of effective prevention and management strategies." @default.
- W2097383660 created "2016-06-24" @default.
- W2097383660 creator A5020103730 @default.
- W2097383660 creator A5059375706 @default.
- W2097383660 date "2011-08-01" @default.
- W2097383660 modified "2023-10-12" @default.
- W2097383660 title "Antiangiogenic agents for the treatment of nonsmall cell lung cancer: characterizing the molecular basis for serious adverse events." @default.
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- W2097383660 doi "https://doi.org/10.3109/07357907.2011.597815" @default.
- W2097383660 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/21740083" @default.
- W2097383660 hasPublicationYear "2011" @default.
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