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- W2097383762 abstract "Abstract We demonstrate that XopD, a type III effector from Xanthomonas campestris pathovar vesicatoria (Xcv), suppresses symptom production during the late stages of infection in susceptible tomato (Solanum lycopersicum) leaves. XopD-dependent delay of tissue degeneration correlates with reduced chlorophyll loss, reduced salicylic acid levels, and changes in the mRNA abundance of senescence- and defense-associated genes despite high pathogen titers. Subsequent structure-function analyses led to the discovery that XopD is a DNA binding protein that alters host transcription. XopD contains a putative helix-loop-helix domain required for DNA binding and two conserved ERF-associated amphiphilic motifs required to repress salicylic acid– and jasmonic acid–induced gene transcription in planta. Taken together, these data reveal that XopD is a unique virulence factor in Xcv that alters host transcription, promotes pathogen multiplication, and delays the onset of leaf chlorosis and necrosis." @default.
- W2097383762 created "2016-06-24" @default.
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- W2097383762 date "2008-07-01" @default.
- W2097383762 modified "2023-10-13" @default.
- W2097383762 title "XopD SUMO Protease Affects Host Transcription, Promotes Pathogen Growth, and Delays Symptom Development in<i>Xanthomonas</i>-Infected Tomato Leaves " @default.
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- W2097383762 doi "https://doi.org/10.1105/tpc.108.058529" @default.
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