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- W2098256851 abstract "from failures in drug supply and from drug intolerance. Shortening of the tuberculosis treatment period for both drugsusceptible and drug-resistant tuberculosis would greatly improve tuberculosis management and infection control. Immunotherapy has been considered as a possible approach toward shortening the duration of chemotherapy, with a wide range of cytokines or their inhibitors and chemical or biological immunomodulatory compounds being explored [2]. However, the factors that determine the replication rate of M. tuberculosis and synergize or antagonize the effectiveness of mycobactericidal drugs remain poorly understood. Efforts at developing new drugs that act on replicating populations of M. tuberculosis, which thrive under the influence of nitric oxide and hypoxia, identified rhodanines [3]. Postchemotherapy relapse was shown to require cyclosporine-sensitive and genetically influenced host immunity, which develops early after infection [4]. Such a relapse can be alleviated by the immunomodulatory agent muramyl dipeptide [5 ]o r by passive antibody, combined with interferon γ (IFN-γ) and anti-interleukin 2 treatment [6]. Recently, an antiinflammatory drug, oxyphenbutazone, was found to be cidal against both replicating and dormant forms of M. tuberculosis [7]. Adjunct treatment for tuberculosis in" @default.
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- W2098256851 date "2013-04-05" @default.
- W2098256851 modified "2023-09-24" @default.
- W2098256851 title "Nonsteroidal Antiinflammatory Drugs for Adjunctive Tuberculosis Treatment" @default.
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- W2098256851 doi "https://doi.org/10.1093/infdis/jit153" @default.
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