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- W2098524307 abstract "CARDIAC allograft vasculopathy or accelerated transplant atherosclerosis is a severe problem for the long-term survival of patients receiving a cardiac allograft. The lesions differ from those of conventional atherosclerosis in several ways and are typically dominated by an excessive concentric myointimal proliferation.1 The known atherogenic factors (tobacco smoking, hypercholesterolemia, etc) probably play a role as well as low-grade rejection episodes. The mechanisms responsible for the development of these lesions are, however, not clear. Many endothelial and macrophage derived cytokines (PDGF, FGF, TGF, etc) have been shown to be growth factors for smooth muscle cells. It has also been shown that oxidized low density lipoprotein (oxLDL) has a mitogenic effect on smooth muscle cells.2 In this work we wanted to study the effect of oxLDL on resting and activated T cells and the possible effects of cytokines produced during alloreaction on smooth muscle cell proliferation." @default.
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- W2098524307 date "1997-08-01" @default.
- W2098524307 modified "2023-10-16" @default.
- W2098524307 title "Oxidized LDL, T lymphocytes, and graft atherosclerosis" @default.
- W2098524307 cites W2077525790 @default.
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- W2098524307 doi "https://doi.org/10.1016/s0041-1345(97)00387-4" @default.
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