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• W2098533214 endingPage "S8" @default.
• W2098533214 startingPage "S1" @default.
• W2098533214 abstract "Resistance to the quinolones almost always arises from the accumulation of mutations in chromosomal genes responsible for the drug targets, permeability, or active efflux. This resistance can be depicted as a stepwise process in which each step, represented by separate mutations, diminishes susceptibility on average 4- to 8-fold. The precise path followed in this stepwise process differs with the quinolone that selects resistance as well as the organism involved. At each step, the influence of each mutation on susceptibility to other quinolones not used in the selection process varies greatly, and a pattern of either cross-resistance or dichotomous resistance may be seen. From an understanding of the stepwise process by which resistance to the quinolones evolves, it is possible to use an 8-fold rule to predict which compounds may provide effective therapy for a given infection and be least likely to select for resistance." @default.
• W2098533214 created "2016-06-24" @default.
• W2098533214 creator A5059871174 @default.
• W2098533214 date "2001-03-15" @default.
• W2098533214 modified "2023-09-24" @default.
• W2098533214 title "Mechanisms Responsible for Cross-Resistance and Dichotomous Resistance among the Quinolones" @default.
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• W2098533214 doi "https://doi.org/10.1086/319369" @default.
• W2098533214 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/11249822" @default.
• W2098533214 hasPublicationYear "2001" @default.
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