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- W2099696651 endingPage "2007" @default.
- W2099696651 startingPage "1997" @default.
- W2099696651 abstract "Polyglutamine diseases are a group of pathologies affecting different parts of the brain and causing dysfunction and atrophy of certain neural cell populations. These diseases stem from mutations in various cellular genes that result in the synthesis of proteins with extended polyglutamine tracts. In particular, this concerns huntingtin, ataxins, and androgen receptor. These mutant proteins can form oligomers, aggregates, and, finally, aggresomes with distinct functions and different degrees of cytotoxicity. In this review, we analyze the effects of different forms of polyQ proteins on other proteins and their functions, which are considered as targets for therapeutic intervention." @default.
- W2099696651 created "2016-06-24" @default.
- W2099696651 creator A5009729836 @default.
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- W2099696651 creator A5079797927 @default.
- W2099696651 creator A5087845947 @default.
- W2099696651 creator A5090053515 @default.
- W2099696651 date "2013-05-15" @default.
- W2099696651 modified "2023-09-26" @default.
- W2099696651 title "Pharmacological protein targets in polyglutamine diseases: Mutant polypeptides and their interactors" @default.
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