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- W2099877518 abstract "Conflicts of interest: none declared. Sir, Erosive vulval lichen planus (LP) is a rare, difficult‐to‐treat inflammatory disease of unknown aetiology. One hypothesis is that a hypersensitivity reaction to a yet unidentified (intraepidermal) antigen may play a role. CD8+ lesional T cells are shown to be more cytotoxic against autologous lesional keratinocytes than clones isolated from nonlesional T‐cell lines; the possible antigen may be associated with major histocompatibility complex class I on lesional keratinocytes.1 In oral LP a hepatitis C‐specific T‐cell response is suggested; however, there is no hint to support this hypothesis for erosive vulval disease in Northern Europe.2 The malignant potential of erosive vulval LP is still a matter of debate. Our objective was to investigate the possibility of an (oncogenic) mucosal human papillomavirus (HPV) being involved in the aetiology and the possible premalignant character of erosive vulval LP. According to the study by Hørding et al., normal vulval skin does not normally harbour HPV DNA (types 6, 11, 16, 18, 33) in women aged 17–98 years (median 64).3 Furthermore, knowledge about the presence of HPV, a potential tumour promoter, in vulval erosive LP is relevant with regard to its treatment. Topical immunosuppressive therapy (tacrolimus or potent glucocorticosteroids) is usually needed, but would be contraindicated if (high‐risk) HPV were present." @default.
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- W2099877518 date "2005-06-01" @default.
- W2099877518 modified "2023-09-26" @default.
- W2099877518 title "No oncogenic mucosal human papillomaviruses in erosive vulval lichen planus" @default.
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- W2099877518 doi "https://doi.org/10.1111/j.1365-2133.2005.06601.x" @default.
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