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• W2100032465 abstract "This study examined the ability of plumbagin to induce apoptosis in chronic myelogenous leukemia (CML). Plumbagin exposure led to a significant reduction in cell viability and the induction of apoptosis. Mechanistically, plumbagin treatment led to elevated levels of ROS. Plumbagin-induced apoptosis was inhibited by N-acetyl L-cysteine (NAC) and PEG-catalase. Furthermore, plumbagin exposure led to elevated expression of DR4 and DR5 and increased killing through soluble TRAIL. The plumbagin-induced increase in DR4 and DR5 was inhibited by treatment with NAC. Together, this study suggests that plumbagin may be an effective treatment of CML through increased sensitivity to TRAIL-mediated killing." @default.
• W2100032465 created "2016-06-24" @default.
• W2100032465 creator A5066113335 @default.
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• W2100032465 date "2011-10-01" @default.
• W2100032465 modified "2023-10-17" @default.
• W2100032465 title "Plumbagin treatment leads to apoptosis in human K562 leukemia cells through increased ROS and elevated TRAIL receptor expression" @default.
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• W2100032465 doi "https://doi.org/10.1016/j.leukres.2011.06.018" @default.
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