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- W2100065398 abstract "<h3>Introduction</h3> Chlorothiazide has been a drug of undisputed value both as a nonmercurial diuretic<sup>1</sup>and as an adjunct in the treatment of systemic hypertension.<sup>2</sup>Side-effects, though uncommon, should nevertheless be recognized in view of the frequent use of this agent and in view of the impression that its use is relatively free of untoward sequelae. In the past few years reported complications of chlorothiazide administration have included thrombocytopenia,<sup>3</sup>agranulocytosis,<sup>4</sup>jaundice,<sup>5</sup>photosensitivity,<sup>6</sup>yellow vision,<sup>7</sup>skin eruptions,<sup>8</sup>hypokalemia,<sup>9,10</sup>pancreatitis,<sup>11, 12A,12B</sup>pancreatic atrophy,<sup>13</sup>and hyperpyruvicacidemia.<sup>14</sup>Of particular interest have been two additional complications: ( 1 ) hyperuricemia<sup>15,16</sup>—alone or with clinical manifestations of gout,<sup>17-20</sup>and (2) hyperglycemia—either in previously nondiabetic subjects or in otherwise well-stabilized diabetics.<sup>21-28</sup>We have recently had an opportunity to observe a patient who, after receiving chlorothiazide continually for four years, developed, in sequence, hyperuricemia with acute" @default.
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- W2100065398 date "1963-04-01" @default.
- W2100065398 modified "2023-09-23" @default.
- W2100065398 title "Chlorothiazide-Induced Gout and Diabetes" @default.
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- W2100065398 doi "https://doi.org/10.1001/archinte.1963.03620280065010" @default.
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