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• W2100083636 abstract "Over the past five decades, per capita caloric intake has increased significantly, and diet- and stress-related diseases are more prevalent. The stress hormone epinephrine stimulates hepatic glucose release during a stress response. The present experiment tested the hypothesis that excess caloric intake alters this ability of epinephrine to increase blood glucose. Sprague–Dawley rats were fed a high-energy cafeteria-style diet (HED). Weight gain during the first 5 days on the diet was used to divide the rats into an HED-lean group and HED-obese group. After 9 weeks, the rats were injected with epinephrine, and blood glucose was measured. HED-obese rats gained body and fat mass, and developed insulin resistance (IR) and hepatic steatosis. HED-lean and control rats did not differ. Epinephrine produced larger increases in blood glucose in the HED-obese rats than in the HED-lean and control rats. Removing the high-energy components of the diet for 4 weeks reversed the potentiated effects of epinephrine on glucose and corrected the IR but not the steatosis or obesity. Consumption of a high-energy cafeteria diet potentiates epinephrine-induced hyperglycemia. This effect is associated with insulin resistance but not adiposity or steatosis and is reversed by 4 weeks of standard chow." @default.
• W2100083636 created "2016-06-24" @default.
• W2100083636 creator A5016129256 @default.
• W2100083636 creator A5064149085 @default.
• W2100083636 creator A5085499494 @default.
• W2100083636 date "2015-04-01" @default.
• W2100083636 modified "2023-09-29" @default.
• W2100083636 title "Excess intake of fat and sugar potentiates epinephrine-induced hyperglycemia in male rats" @default.
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• W2100083636 doi "https://doi.org/10.1016/j.jdiacomp.2014.12.017" @default.
• W2100083636 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25716573" @default.
• W2100083636 hasPublicationYear "2015" @default.
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