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• W2100084881 endingPage "1232" @default.
• W2100084881 startingPage "1219" @default.
• W2100084881 abstract "The obligate intracellular bacterial pathogen Chlamydia trachomatis is a major cause of blindness and sexually transmitted diseases. Like the enteric pathogens Salmonella and Shigella, Chlamydia injects effector proteins into epithelial cells to initiate extensive remodeling of the actin cytoskeleton at the bacterial attachment site, which culminates in the engulfment of the bacterium by plasma membrane extensions. Numerous Salmonella and Shigella effectors promote this remodeling by activating Rho GTPases and tyrosine kinase signaling cascades and by directly manipulating actin dynamics. Recent studies indicate that similar host-cell alterations occur during Chlamydia invasion, but few effectors are known. The identification of additional Chlamydia effectors and the elucidation of their modes of function are critical steps towards an understanding of how this clinically important pathogen breaches epithelial surfaces and causes infection." @default.
• W2100084881 created "2016-06-24" @default.
• W2100084881 creator A5021498852 @default.
• W2100084881 creator A5089779586 @default.
• W2100084881 date "2010-08-01" @default.
• W2100084881 modified "2023-10-16" @default.
• W2100084881 title "Uncivil engineers:<i>Chlamydia</i>,<i>Salmonella</i>and<i>Shigella</i>alter cytoskeleton architecture to invade epithelial cells" @default.
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