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• W2100085583 abstract "Cardiotrophin-like cytokine (CLC), also known as novel neurotrophin-1/B cell stimulating factor-3 (NNT-1/BSF-3), is a recently identified member of the interleukin (IL)-6 family of cytokines that share gp130 as a signal-transducing receptor component. In this study, we demonstrate that CLC is expressed in fetal mouse neuroepithelial cells and has a potential to induce their astrocyte differentiation in a synergistic manner with bone-morphogenetic protein (BMP)-2, which is also expressed in the fetal mouse brain. CLC-stimulation led to promoter activation of the gene for an astrocyte marker, glial fibrillary acidic protein (GFAP), which was clearly inhibited by expression of a dominant negative form of a transcription factor, STAT3, or by introduction of a mutation in a single STAT3-binding site in the promoter, suggesting a critical role of STAT3 in the CLC-induced GFAP transcription. These results suggest that CLC plays a role in astrocyte differentiation via STAT3 activation within the developing brain." @default.
• W2100085583 created "2016-06-24" @default.
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• W2100085583 date "2002-04-01" @default.
• W2100085583 modified "2023-10-14" @default.
• W2100085583 title "CARDIOTROPHIN-LIKE CYTOKINE INDUCES ASTROCYTE DIFFERENTIATION OF FETAL NEUROEPITHELIAL CELLS VIA ACTIVATION OF STAT3" @default.
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• W2100085583 doi "https://doi.org/10.1006/cyto.2002.1006" @default.
• W2100085583 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/12090754" @default.
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